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Cerebral Proliferative Angiopathy

Published online by Cambridge University Press:  02 December 2014

Muneer Eesa*
Affiliation:
Department of Diagnostic Imaging, Foothills Medical Centre, Calgary, Alberta, Canada
Pranshu Sharma
Affiliation:
Department of Diagnostic Imaging, Foothills Medical Centre, Calgary, Alberta, Canada
Mayank Goyal
Affiliation:
Department of Diagnostic Imaging, Foothills Medical Centre, Calgary, Alberta, Canada
*
Department of Diagnostic Imaging, Foothills Medical Centre, 1403 29th Street NW, Calgary, Alberta, T2N 2T9, Canada
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A 31-year-old male presented with transient numbness of the right hand, progressing to involve the forearm. The symptoms lasted for around 30 minutes without any associated weakness or positive motor phenomena. An evaluation in the emergency room revealed no focal deficits.Acomputer tomogram (CT) scan performed in the emergency room suggested a few abnormal vascular structures in relation to the left hemisphere, with no evidence of hemorrhage. The patient was referred for magnetic resonance imaging (MRI) and conventional angiography.

The MRI showed a diffuse network of densely enhancing vascular spaces involving the left middle cerebral artery (MCA) and anterior cerebral artery (ACA) territories without a clear nidus or abnormal parenchymal signal change (Figure 1). There was no involvement of the basal ganglia or thalami. Given the size and extent of the abnormality, there was a relative paucity of draining vessels, which were only moderately enlarged.

Type
Neuroimaging Highlight
Copyright
Copyright © The Canadian Journal of Neurological 2009

References

1.Wallace, RC, Bourekas, EC.Brain arteriovenous malformations. Neuroimaging Clin N Am. 1998;8:38399.Google Scholar
2.Lasjaunias, PL, Landrieu, P, Rodesch, G, Alvarez, H, Ozanne, A, Holmin, S., et al.Cerebral proliferative angiopathy: clinical and angiographic description of an entity different from cerebral AVMs. Stroke. 2008;39:87885.Google Scholar
3.Chin, LS, Raffel, C, Gonzalez-Gomez, I, Giannotta, SL, McComb, JG.Diffuse arteriovenous malformations: a clinical, radiological, and pathological description. Neurosurgery. 1992;31:8638; discussion 8689.Google Scholar
4.Mulliken, JB, Glowacki, J.Hemangiomas and vascular malformations in infants and children: a classification based on endothelial characteristics. Plast Reconstr Surg. 1982;69:41222.Google Scholar
5.Berenstein, A, Lasjaunias, PL, TerBrugge, KG.Surgical neuroangiography, Volume 2, 2nd ed. Heidelberg, Berlin: Springer Verlag; 2004.Google Scholar
6.Ducreux, D, Meder, JF, Fredy, D, Bittoun, J, Lasjaunias, P.MR perfusion imaging in proliferative angiopathy. Neuroradiology. 2004;46:10512.Google Scholar