Hostname: page-component-848d4c4894-hfldf Total loading time: 0 Render date: 2024-06-01T23:30:38.523Z Has data issue: false hasContentIssue false

The Influence of Avitaminosis on the Course of Trypanosome Infection

Published online by Cambridge University Press:  15 May 2009

J. Fine
Affiliation:
Usher Institute of Public Health, Edinburgh University
Rights & Permissions [Opens in a new window]

Extract

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

Recent research has shown that the term “anti-infective” applied to vitamin A is misleading and inaccurate. Harris (1933) has discussed the evidence which shows that vitamin A cannot be regarded as a general antiinfective agent: the actual relation of the vitamin to infection has been well defined by Cramer, who states (1930) that “an adequate supply of vitamin A is a powerful prophylactic against infections entering by mucous membranes, but there is no evidence that it can prevent or cure those infections which enter by the blood stream or by subcutaneous tissues.”

Type
Research Article
Copyright
Copyright © Cambridge University Press 1934

References

REFERENCES

Burton, and Burton, (1930). Lancet, 1, 1063.Google Scholar
Cramer, (1923). Lancet 1, 1046.Google Scholar
Cramer, (1930). Lancet 1, 1153.Google Scholar
Drummond, (1919). Bioch. J. 13, 91.Google Scholar
Green, and Mellanby, (1928). Brit. Med. J. 2, 691.CrossRefGoogle Scholar
Harris, L. (1933). Brit. Med. J. 2, 231.CrossRefGoogle Scholar
Harris, and Griffiths, (1932). Lancet, 2, 616.Google Scholar
Pryde, (1928). Recent Advances in Biochemistry, p. 249.Google Scholar