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9 - Evaluation of speech and language problems

from Part I - Speech and language problems

Published online by Cambridge University Press:  26 October 2009

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Summary

Much of learning and the day-to-day social interactions of life depend on communication. Subtle speech and language problems impair learning and inhibit social development. Discipline, explanations, and instructions are based largely on the spoken word.

Subtle seizure short-circuits may impair the epileptic child's functioning in many ways. Proper help depends on early recognition and confirmation of the problem, followed by appropriate, practical, remediative help. Children who have epilepsy are at risk of developing speech and language problems and should be evaluated at four levels, depending on their needs: (1) awareness and subsequent screening, (2) formal basic hearing and speech testing, (3) in-depth language-processing studies, as indicated, and (4) applied language arts, especially in practical situations, by both the therapist and others (parents, teachers, etc.). The main challenge is suspecting the problem and confirming that it exists (Bradford, 1980).

Too often, a physician tells the parents that a young child will probably grow out of the problem. This is not true. A child who has delayed speech or a deviation from normal that is significant enough to disturb the parents, frustrate the child, or be apparent to the teacher or physician should be referred for speech and language evaluation. Both relaxed testing and testing under stress is helpful, as stress can bring out deficits that may be overlooked in the usual, relaxed speech-testing situation.

Awareness and screening

Language problems in children with epilepsy occur frequently enough such that all children with epilepsy should be screened for possible speech and language problems.

Type
Chapter
Information
Childhood Epilepsy
Language, Learning and Behavioural Complications
, pp. 133 - 140
Publisher: Cambridge University Press
Print publication year: 2004

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References

Aktekin, B., Ozkaynak, S., Oguz, Y., et al. (1999) Short term effects of antiepileptic drugs on P300 in patients with epilepsy. 23rd International Epilepsy Congress.Epilepsia 40 (suppl 2): 124Google Scholar
Armon, C., Paul, R. G., Miller, P., et al. (1991). Valproate induced hearing impairment: active ascertainment in an epilepsy clinic population and result of therapeutic intervention.Epilepsia 32 (suppl 3): 8Google Scholar
Bradford, L. J. (1980). Understanding and assessing communicative disorders in children. J. Dev. Behav. Pediatr. 1: 89–95CrossRefGoogle ScholarPubMed
Haan, J. & Schulz, G. A. (1991). Cognitive evoked potentials in complex partial and generalized epilepsies. 19th International Epilepsy Congress. Epilepsia 32 (suppl 1): 117Google Scholar
Karageorgiou, C., Kontogianni, B. & Tagaris, G. (1991). Brainstem auditory evoked potentials in epileptics patients receiving long-term valproate monotherapy. 19th International Epilepsy Congress. Epilepsia 32 (suppl 1): 27Google Scholar
Mervaala, E., Kalviainen, R., Kjnononen, M., et al. (1993). Vigabatrin and carbamazepine monotherapy does not impair cognitive functions in event-related potentials.Epilepsia 34 (suppl 6): 95Google Scholar
Morales, A., Verhulst, S., Faingold, C. L., et al. (1992). Absence epilepsy: brainstem auditory evoked responses (BAERs) before and during treatment.Epilepsia 33 (suppl 3): 63Google Scholar
Syrigou-Papavasiliou, A., LeWitt, P. A., Green, V., et al. (1985). P300 and temporal lobe epilepsy.Epilepsia 26: 528Google Scholar

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