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Adrenocortical Responsiveness during Courses of Electro-convulsive Therapy

Published online by Cambridge University Press:  29 January 2018

A. Elithorn ,
P. K. Bridges ,
J. R. Hodges  and
M. T. Jones
A. Elithorn
Affiliation:
Department of Psychological Medicine, Royal Free Hospital, London, W.C.1
P. K. Bridges
Affiliation:
Department of Psychological Medicine, Royal Free Hospital, London, W.C.1
J. R. Hodges
Affiliation:
Department of Pharmacology, Royal Free Hospital Medical School
M. T. Jones
Affiliation:
Department of Pharmacology, Royal Free Hospital Medical School
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Extract

In a previous paper (Hodges, Jones, Elithorn and Bridges, 1964) we reported on adrenocortical activity in depressed and schizophrenic patients as revealed by plasma cortisol levels before and after electro-convulsive therapy (E.C.T.). Close similarity was found between the two groups except for three depressed patients who appeared to show considerably higher cortisol levels after the treatment than did the remaining subjects. The patients were examined at random different treatments during the whole treatment course and it appeared possible, both that the observed cortisol response to E.C.T. might depend partly on which treatment of the series in a whole course was under examination, and also that the response of the illness to therapy might be a significant factor. It was therefore decided to observe in a number of subjects the response to successive treatments throughout courses of E.C.T.

Type
Biochemical Studies
Information
The British Journal of Psychiatry , Volume 115 , Issue 522 , May 1969 , pp. 575 - 580
Copyright
Copyright © Royal College of Psychiatrists, 1969 

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References

Bliss, E. L., Migeon, G. J., Branch, C. H. H., and Samuels, L. T. (1955). Amer. J. Psychiat., 112, 358.Google Scholar
Blumberg, A. G. (1960). Psychosom. Med., 22, 32.Google Scholar
Board, F., Wadeson, R., and Persky, H. (1957). Arch. Neurol. Psychiat. (Chic.), 78, 612.Google Scholar
Bridges, P. K., and Jones, M. T. (1966). Brit. J. Psychiat., 112, 1257.Google Scholar
Co Tui, F., Brinitzer, W., and Orr, A. (1961). Psychiat. Quart., 35, 432.CrossRefGoogle Scholar
Cope, C. L. (1965). Adrenal Steroids and Disease. Pitman Medical, London, p. 34.Google Scholar
Eiduson, S., Brill, N. Q., and Crumpton, E. (1961). Arch. gen. Psychiat. (Chic.), 5, 227.Google Scholar
Elithorn, A., Piercy, M. F., and Crosskey, M. A. (1955). J. Neurol. Neurosurg. Psychiat., 18, 34.Google Scholar
Gibbons, J. L., and McHugh, P. R. (1962). J. psychiat. Res., 1, 162.CrossRefGoogle Scholar
Glaser, G. H. (1952). J. nerv. ment. Dis., 115, 189.Google Scholar
Hamilton, M., and White, J. M. (1960). Brit. J. Psychiat., 106, 1031.Google Scholar
Hartley, M. O. (1961). Technometrics, 3, 269.CrossRefGoogle Scholar
Hodges, J. R., Jones, M., Eltthorn, A., and Bridges, P. (1964). Nature, 204, 754.Google Scholar
Mittelman, A., Romanoff, L. P., Pincus, G., and Hoagland land, H. J. (1952). J. clin. Endocr., 12, 831.Google Scholar
Pincus, G., and Hoagland, H. (1950). Amer. J. Psychiat., 106, 641.Google Scholar
Stemmermann, M. G., and Owen, T. V. (1957). J. clin. exp. Psychopath., 18, 236.Google Scholar
Zenker, N., and Bernstein, D. E. (1958). J. biol. Chem., 231, 695.Google Scholar
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