Skip to main content Accessibility help
×
Hostname: page-component-77c89778f8-gq7q9 Total loading time: 0 Render date: 2024-07-22T22:30:45.210Z Has data issue: false hasContentIssue false

Chapter 3 - Early Pregnancy Loss with Abnormal Karyotype

from Section 2 - Early Pregnancy Pathology

Published online by Cambridge University Press:  03 September 2018

Raymond W. Redline
Affiliation:
Case Western Reserve University, Ohio
Theonia K. Boyd
Affiliation:
Harvard Medical School, Boston
Drucilla J. Roberts
Affiliation:
Harvard Medical School, Boston
Get access
Type
Chapter
Information
Publisher: Cambridge University Press
Print publication year: 2017

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Committee on Practice B-G. The American College of Obstetricians and Gynecologists Practice Bulletin no. 150. Early pregnancy loss. Obstet Gynecol. 2015;125:1258–67.Google Scholar
Hassold, T, Abruzzo, M, Adkins, K, et al. Human aneuploidy: incidence, origin, and etiology. Environ Mol Mutagen. 1996;28:167–75.Google Scholar
Farquaharson, HG, Stephenson, MD. Early pregnancy: Cambridge University Press; 2010.Google Scholar
Redline, RW, Zaragoza, MV, Hassold, T. Prevalence of developmental and inflammatory lesions in non-molar first trimester spontaneous abortions. Hum Pathol. 1999;30:93100.Google Scholar
Rushton, DI. The classification and mechanisms of spontaneous abortion. Perspect Pediatr Pathol. 1984;8:269–87.Google Scholar
Zaragoza, MV, Surti, U, Redline, RW, et al. Origin and phenotype of triploidy in spontaneous abortions: predominance of diandry and its association with partial hydatidiform mole. Am J Hum Genet. 2000;66:1807–20.Google Scholar
Lage, JM, Bagg, A. Hydatidiform moles: DNA flow cytometry, image analysis and selected topics in molecular biology. Histopathology. 1996;28:379–82.Google Scholar
Salafia, C, Maier, D, Vogel, C, et al. Placental and decidual histology in spontaneous abortion:Detailed description and correlations with chromosome number. Obstet Gynecol. 1993;82:295303.Google Scholar
Novak, R, Agamanolis, D, Dasu, S, et al. Histologic analysis of placental tissue in first trimester abortions. Pediatr Pathol. 1988;8:477–82.Google Scholar
Genest, DR, Roberts, D, Boyd, T, et al. Fetoplacental histology as a predictor of karyotype: a controlled study of spontaneous first trimester abortions. Hum Pathol. 1995;26:201–9.Google Scholar
van Lijnschoten, G, Arends, JW, De La Fuente, AA, et al. Intra- and inter-observer variation in the interpretation of histological features suggesting chromosomal abnormality in early abortion specimens. Histopathology. 1993;22:25–9.Google Scholar
Jindal, P, Regan, L, Fourkala, EO, et al. Placental pathology of recurrent spontaneous abortion: the role of histopathological examination of products of conception in routine clinical practice: a mini review. Hum Reprod. 2007;22:313–6.Google Scholar
Dillon, J, Gonzalez, JL, Parks, WT, et al. Frequent aneuploidy detection in non-molar conceptuses by molecular analysis of products of conception with atypical villus morphology. Mod Pathol. 2017;30 Suppl 2:282A.Google Scholar
Redline, RW, Hassold, T, Zaragoza, MV. Determinants of trophoblast hyperplasia in spontaneous abortions. Mod Pathol. 1998;11:762–8.Google Scholar
Redline, RW, Hassold, T, Zaragoza, MV. Prevalence of the partial molar phenotype in triploidy of maternal and paternal origin. Hum Pathol. 1998;28:505–11.Google Scholar
Kliman, HJ, Milano, KM. The majority of miscarriages are caused by genetic abnormalities. Fertil Steril. 2013;100 (3):S306.CrossRefGoogle Scholar
Kliman, HJ, Segel, L. The placenta may predict the baby. J Theor Biol. 2003;225:143–5.Google Scholar
Ornoy, A, Salamon-Arnon, J, Ben-Zur, Z, et al. Placental findings in spontaneous abortions and stillbirths. Teratology. 1981;24:243–52.Google Scholar
Klausen, S, Larsen, LG. Partial moles with maze-like vascular anomaly. APMIS. 1994;102:638–40.Google Scholar
Zaragoza, MV, Millie, E, Redline, RW, et al. Studies of nondysjunction in trisomies 2, 7, 15, and 22: does the parental origin of trisomy influence placental morphology. J Med Genet. 1998;35:924–31.CrossRefGoogle Scholar

Save book to Kindle

To save this book to your Kindle, first ensure coreplatform@cambridge.org is added to your Approved Personal Document E-mail List under your Personal Document Settings on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part of your Kindle email address below. Find out more about saving to your Kindle.

Note you can select to save to either the @free.kindle.com or @kindle.com variations. ‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi. ‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.

Find out more about the Kindle Personal Document Service.

Available formats
×

Save book to Dropbox

To save content items to your account, please confirm that you agree to abide by our usage policies. If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account. Find out more about saving content to Dropbox.

Available formats
×

Save book to Google Drive

To save content items to your account, please confirm that you agree to abide by our usage policies. If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account. Find out more about saving content to Google Drive.

Available formats
×